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CCT - Certified Cardiographic Technician 12-Lead ECG Interpretation and Cardiac Conditions Questions and Answers Flashcards

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  1. True posterior MI is best confirmed on a 12-lead ECG by which finding in the standard leads?

    Answer: Tall broad R waves and ST depression in V1-V2 (mirror of posterior STEMI)

    Posterior MI appears as a mirror image in V1-V2: tall, broad R waves (posterior Q equivalents) and ST depression (reciprocal of posterior ST elevation), confirmed with posterior leads V7-V9.

  2. A patient's 12-lead ECG shows a short PR interval (<120 ms), delta wave, and wide QRS. This triad defines:

    Answer: Wolff-Parkinson-White (WPW) syndrome pre-excitation pattern

    The WPW pre-excitation pattern consists of a short PR interval (accessory pathway bypasses AV node), a delta wave (initial slow ventricular activation via the pathway), and a widened QRS.

  3. Which lead is considered the best single-lead view for detecting atrial activity and identifying P-wave morphology?

    Answer: V1

    Lead V1 sits directly over the right atrium and provides the clearest biphasic P wave, making it ideal for identifying atrial activity, flutter waves, and P-wave morphology.

  4. On a 12-lead ECG, a Brugada pattern is most commonly seen in which lead grouping?

    Answer: Right precordial leads V1-V2

    Brugada pattern (coved-type ST elevation with RBBB morphology) is characteristically seen in the right precordial leads V1 and V2, reflecting right ventricular outflow tract abnormality.

  5. Left axis deviation (LAD) on a 12-lead ECG is defined as the QRS axis falling between:

    Answer: -30° and -90°

    Left axis deviation is defined as a QRS axis more negative than -30°, with the range of -30° to -90° representing pathological LAD on a standard 12-lead ECG.

  6. Which finding on a 12-lead ECG is most specific for an acute anterior STEMI caused by proximal LAD occlusion?

    Answer: ST elevation in V1-V4 with reciprocal depression in II, III, aVF

    Anterior STEMI from proximal LAD occlusion causes ST elevation in V1-V4 with reciprocal inferior ST depression in II, III, and aVF, indicating a large ischemic territory.